List of cutaneous conditions

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Young child with a red rash covering face, chest, shoulders, and arms
Measles

Many conditions affect the human integumentary system—the organ system covering the entire surface of the body and composed of skin, hair, nails, and related muscle and glands.[1] The major function of this system is as a barrier against the external environment.[2] The skin weighs an average of four kilograms, covers an area of two square meters, and is made of three distinct layers: the epidermis, dermis, and subcutaneous tissue.[1] The two main types of human skin are: glabrous skin, the hairless skin on the palms and soles (also referred to as the "palmoplantar" surfaces), and hair-bearing skin.[3] Within the latter type, the hairs occur in structures called pilosebaceous units, each with hair follicle, sebaceous gland, and associated arrector pili muscle.[4] In the embryo, the epidermis, hair, and glands form from the ectoderm, which is chemically influenced by the underlying mesoderm that forms the dermis and subcutaneous tissues.[5][6][7]

The epidermis is the most superficial layer of skin, a squamous epithelium with several strata: the stratum corneum, stratum lucidum, stratum granulosum, stratum spinosum, and stratum basale.[8] Nourishment is provided to these layers by diffusion from the dermis, since the epidermis is without direct blood supply. The epidermis contains four cell types: keratinocytes, melanocytes, Langerhans cells, and Merkel cells. Of these, keratinocytes are the major component, constituting roughly 95 percent of the epidermis.[3] This stratified squamous epithelium is maintained by cell division within the stratum basale, in which differentiating cells slowly displace outwards through the stratum spinosum to the stratum corneum, where cells are continually shed from the surface.[3] In normal skin, the rate of production equals the rate of loss; about two weeks are needed for a cell to migrate from the basal cell layer to the top of the granular cell layer, and an additional two weeks to cross the stratum corneum.[9]

The dermis is the layer of skin between the epidermis and subcutaneous tissue, and comprises two sections, the papillary dermis and the reticular dermis.[10] The superficial papillary dermis interdigitates with the overlying rete ridges of the epidermis, between which the two layers interact through the basement membrane zone.[10] Structural components of the dermis are collagen, elastic fibers, and ground substance.[10] Within these components are the pilosebaceous units, arrector pili muscles, and the eccrine and apocrine glands.[8] The dermis contains two vascular networks that run parallel to the skin surface—one superficial and one deep plexus—which are connected by vertical communicating vessels.[8][11] The function of blood vessels within the dermis is fourfold: to supply nutrition, to regulate temperature, to modulate inflammation, and to participate in wound healing.[12][13]

The subcutaneous tissue is a layer of fat between the dermis and underlying fascia.[14] This tissue may be further divided into two components, the actual fatty layer, or panniculus adiposus, and a deeper vestigial layer of muscle, the panniculus carnosus.[3] The main cellular component of this tissue is the adipocyte, or fat cell.[14] The structure of this tissue is composed of septal (i.e. linear strands) and lobular compartments, which differ in microscopic appearance.[8] Functionally, the subcutaneous fat insulates the body, absorbs trauma, and serves as a reserve energy source.[14]

Conditions of the human integumentary system constitute a broad spectrum of diseases, also known as dermatoses, as well as many nonpathologic states (like, in certain circumstances, melanonychia and racquet nails).[15][16] While only a small number of skin diseases account for most visits to the physician, thousands of skin conditions have been described.[14] Classification of these conditions often presents many nosological challenges, since underlying etiologies and pathogenetics are often not known.[17][18] Therefore, most current textbooks present a classification based on location (for example, conditions of the mucous membrane), morphology (chronic blistering conditions), etiology (skin conditions resulting from physical factors), and so on.[19][20] Clinically, the diagnosis of any particular skin condition is made by gathering pertinent information regarding the presenting skin lesion(s), including the location (such as arms, head, legs), symptoms (pruritus, pain), duration (acute or chronic), arrangement (solitary, generalized, annular, linear), morphology (macules, papules, vesicles), and color (red, blue, brown, black, white, yellow).[21] Diagnosis of many conditions often also requires a skin biopsy which yields histologic information[22][23] that can be correlated with the clinical presentation and any laboratory data.[24][25][26]

Acneiform eruptions

Acneiform eruptions are caused by changes in the pilosebaceous unit.[27][28]

Autoinflammatory syndromes

Autoinflammatory syndromes are a group of inherited disorders characterized by bouts of inflammatory skin lesions and periodic fevers.[29][30]

Chronic blistering

Chronic blistering cutaneous conditions have a prolonged course and present with vesicles and bullae.[31][32][33]

Conditions of the mucous membranes

Conditions of the mucous membranes involve the moist linings of the eyes, nose, mouth, genitals, and anus.[34]

Conditions of the skin appendages

Conditions of the skin appendages are those affecting the glands of the skin, hair, nails, and arrector pili muscles.[1][35]

Conditions of the subcutaneous fat

Conditions of the subcutaneous fat are those affecting the layer of adipose tissue that lies between the dermis and underlying fascia.[36][37][38][39]

Congenital anomalies

Cutaneous congenital anomalies are a diverse group of disorders that result from faulty morphogenesis, the biological process that forms the shape of a human body.[35][40][41]

Connective tissue diseases

Connective tissue diseases are caused by a complex array of autoimmune responses that target or affect collagen or ground substance.[35][42]

Abnormalities of dermal fibrous and elastic tissue

Abnormalities of dermal fibrous and elastic tissue are caused by problems in the regulation of collagen synthesis and/or degradation.[35][43]

Fuzzy red lines on an abdomen
Striae distensae

Dermal and subcutaneous growths

Dermal and subcutaneous growths result from (1) reactive or neoplastic proliferation of cellular components of the dermis or subcutaneous tissue, or (2) neoplasms invading or aberrantly present in the dermis.[1][35]

Dermatitis

Dermatitis is a general term for "inflammation of the skin".[44]

Atopic

Atopic dermatitis is a chronic dermatitis associated with a hereditary tendency to develop allergies to food and inhalant substances.[45][46][47]

Contact

Contact dermatitis is caused by certain substances coming in contact with the skin.[48][49][50]

Eczema

Eczema refers to a broad range of conditions that begin as spongiotic dermatitis and may progress to a lichenified stage.[26][51]

Small blisters and crusting on the distal fingertips
Dyshidrosis

Pustular

Pustular dermatitis is an inflammation of the skin that presents with pustular lesions.[26][52]

Seborrheic

Seborrheic dermatitis is a chronic, superficial, inflammatory disease characterized by scaling on an erythematous base.[53]

Disturbances of pigmentation

Disturbances of human pigmentation, either loss or reduction, may be related to loss of melanocytes or the inability of melanocytes to produce melanin or transport melanosomes correctly.[54][55][56]

Drug eruptions

Drug eruptions are adverse drug reactions that present with cutaneous manifestations.[57][58][59]

Endocrine conditions often present with cutaneous findings as the skin interacts with the endocrine system in many ways.[60][61]

Eosinophilic

Eosinophilic cutaneous conditions encompass a wide variety of diseases that are characterized histologically by the presence of eosinophils in the inflammatory infiltrate, or evidence of eosinophil degranulation.[62][63]

Epidermal nevi, neoplasms, cysts

Epidermal nevi, neoplasms, cysts are skin lesions that develop from the epidermal layer of the skin.[8][26]

Erythemas

Large erythematous patch in the pattern of a "bull's-eye" on a woman's posterior upper arm
Erythema migrans

Erythemas are reactive skin conditions in which there is blanchable redness.[1][9]

Genodermatoses

Genodermatoses are inherited genetic skin conditions often grouped into three categories: chromosomal, single gene, and polygenetic.[64][65]

See also: Category:Infection-related cutaneous conditions and Skin and skin structure infection

Infection-related cutaneous conditions may be caused by bacteria, fungi, yeast, viruses, and/or parasites.[26][66]

See also: Category:Bacterium-related cutaneous conditions and Bacterial skin infection

Bacterium-related cutaneous conditions often have distinct morphologic characteristics that may be an indication of a generalized systemic process or simply an isolated superficial infection.[66][67]

Mycobacterium-related cutaneous conditions are caused by Mycobacterium infections.[66][68]

See also: Category:Mycosis-related cutaneous conditions and Fungal skin infection

Mycosis-related cutaneous conditions are caused by fungi or yeasts, and may present as either a superficial or deep infection of the skin, hair, and/or nails.[66]

Parasitic infestations, stings, and bites

Parasitic infestations, stings, and bites in humans are caused by several groups of organisms belonging to the following phyla: Annelida, Arthropoda, Bryozoa, Chordata, Cnidaria, Cyanobacteria, Echinodermata, Nemathelminthes, Platyhelminthes, and Protozoa.[66][69]

See also: Category:Virus-related cutaneous conditions and Viral skin infections

Virus-related cutaneous conditions are caused by two main groups of viruses–DNA and RNA types–both of which are obligatory intracellular parasites.[66][70]

Lichenoid eruptions

Lichenoid eruptions are dermatoses related to the unique, common inflammatory disorder lichen planus, which affects the skin, mucous membranes, nails, and hair.[71][72][73]

Lymphoid-related cutaneous conditions are a group of disorders characterized by collections of lymphocyte cells within the skin.[74]

Melanocytic nevi and neoplasms

Melanocytic nevi and neoplasms are caused by either a proliferation of (1) melanocytes, or (2) nevus cells, a form of melanocyte that lack dendritic processes.[75][76]

Melanoma

Melanoma is a malignant proliferation of melanocytes and the most aggressive type of skin cancer.[77][78][79]

Monocyte- and macrophage-related

Monocyte- and macrophage-related cutaneous conditions are characterized histologically by infiltration of the skin by monocyte and/or macrophage cells,[10] often divided into several categories, including granulomatous disease,[80] histiocytoses,[81] and sarcoidosis.[82]

Mucinoses

Mucinoses are a group of conditions caused by dermal fibroblasts producing abnormally large amounts of mucopolysaccharides.[34]

Neurocutaneous

Neurocutaneous conditions are due organic nervous system disease or are psychiatric in etiology.[83][84]

Noninfectious immunodeficiency-related cutaneous conditions are caused by T-cell and/or B-cell dysfunction.[85][86]

Nutrition-related cutaneous conditions are caused by malnutrition due to an improper or inadequate diet.[87][88]

Papulosquamous hyperkeratotic

Papulosquamous hyperkeratotic cutaneous conditions are those that present with papules and scales caused by a thickening of the stratum corneum.[9]

Multiple, red, well demarcated papules and plaques on the flank of an adult male
Pityriasis rosea

Palmoplantar keratodermas

Palmoplantar keratodermas are a diverse group of hereditary and acquired keratodermas in which there is hyperkeratosis of the skin of the palms and soles.[89]

Bottom of an adult's foot with a thickened and scaling surface
Palmoplantar keratoderma
Linear, vertical brown patch inferior to umbilicus on the abdomen of a pregnant woman
Linea nigra

Pregnancy-related cutaneous conditions are a group of skin changes observed during pregnancy.[90][91]

Pruritic

Pruritus, commonly known as itchiness, is a sensation exclusive to the skin, and characteristic of many skin conditions.[92][93]

Psoriasis

Psoriasis is a common, chronic, and recurrent inflammatory disease of the skin characterized by circumscribed, erythematous, dry, scaling plaques.[94][95][96]

Large, red, scaly plaque
Psoriasis vulgaris

Reactive neutrophilic

Reactive neutrophilic cutaneous conditions constitute a spectrum of disease mediated by neutrophils, and typically associated with underlying diseases, such as inflammatory bowel disease and hematologic malignancy.[97][98]

Multiple ulcerations with undermined edges on the adult back and upper, posterior arm
Pyoderma gangrenosum

Recalcitrant palmoplantar eruptions

Recalcitrant palmoplantar eruptions are skin conditions of the palms and soles which are resistant to treatment.[34]

Resulting from errors in metabolism

Skin conditions resulting from errors in metabolism are caused by enzymatic defects that lead to an accumulation or deficiency of various cellular components, including, but not limited to, amino acids, carbohydrates, and lipids.[16]

Resulting from physical factors

Skin conditions resulting from physical factors occur from a number of causes, including, but not limited to, hot and cold temperatures, friction, and moisture.[34][99][100]

Ionizing radiation-induced

Ionizing radiation-induced cutaneous conditions result from exposure to ionizing radiation.[101]

Urticaria and angioedema

Urticaria is a vascular reaction of the skin characterized by the appearance of wheals, which are firm, elevated swelling of the skin.[102] Angioedema, which can occur alone or with urticaria, is characterized by a well-defined, edematous swelling that involves subcutaneous tissues, abdominal organs, and/or upper airway.[103]

Raised, edematous, red skin lesions on the abdomen
Acute urticaria

Vascular-related cutaneous conditions result from dysfunction of the blood and/or blood vessels in the dermis, or lymphatics in the subcutaneous tissues.[9][104][105]

Footnotes

  1. ↑ Any given cutaneous condition is only included once within this list.
  2. ↑ Parentheticals are used to indicate other names by which a condition is known. If there are multiple alternative names for a condition, they are separated by commas within the parenthetical.
  3. ↑ Citations for any given condition name and/or alternative name(s) may be found within the condition's respective article.
  4. ↑ This list uses American English; therefore, the symbols æ and œ, which are common to British English, are not used, but, rather, simplified to a single e. For example, the spelling of nevus is favored over nævus, edema over œdema, and so forth. For more information, see American and British English differences.
  5. ↑ Non-English names are included within this list when those terms are found in English medical literature. Inclusion of acne excoriée des jeunes filles (French), Frambösie (German), and parangi (Malay) represent examples of this convention.
  6. ↑ Abbreviations for condition names commonly described in medical literature with an acronym or initialism are included within this list.
  7. ↑ Within this list, the term immunoglobulin is abbreviated to Ig when used as a prefix to a specific antibody isotype (i.e. IgA, IgD, IgE, IgG, and IgM).
  8. ↑ Within this list, the terms human immunodeficiency virus and acquired immunodeficiency syndrome are abbreviated to HIV and AIDS, respectively.

See also

References

  1. 1 2 3 4 5 Miller, Jeffrey H; Marks, James G (2006). Lookingbill and Marks' Principles of Dermatology. Saunders. ISBN 1-4160-3185-5.
  2. ↑ Lippens S, Hoste E, Vandenabeele P, Agostinis P, Declercq W; Hoste; Vandenabeele; Agostinis; Declercq (April 2009). "Cell death in the skin". Apoptosis. 14 (4): 549–69. doi:10.1007/s10495-009-0324-z. PMID 19221876.
  3. 1 2 3 4 Burns, Tony; et al. (2006) Rook's Textbook of Dermatology CD-ROM. Wiley-Blackwell. ISBN 1-4051-3130-6.
  4. ↑ Paus R, Cotsarelis G; Cotsarelis (1999). "The biology of hair follicles". N Engl J Med. 341 (7): 491–7. doi:10.1056/NEJM199908123410706. PMID 10441606.
  5. ↑ Goldsmith, Lowell A. (1983). Biochemistry and physiology of the skin. Oxford University Press. ISBN 0-19-261253-0.
  6. ↑ Fuchs E (February 2007). "Scratching the surface of skin development". Nature. 445 (7130): 834–42. Bibcode:2007Natur.445..834F. doi:10.1038/nature05659. PMC 2405926Freely accessible. PMID 17314969.
  7. ↑ Fuchs E, Horsley V; Horsley (April 2008). "More than one way to skin .". Genes Dev. 22 (8): 976–85. doi:10.1101/gad.1645908. PMC 2732395Freely accessible. PMID 18413712.
  8. 1 2 3 4 5 Freedberg, Irwin M;; et al. (2003). Fitzpatrick's Dermatology in General Medicine. McGraw-Hill. ISBN 0-07-138076-0.
  9. 1 2 3 4 Bolognia, Jean L;; et al. (2007). Dermatology. St. Louis: Mosby. ISBN 1-4160-2999-0.
  10. 1 2 3 4 Rapini, Ronald P (2005). Practical dermatopathology. Elsevier Mosby. ISBN 0-323-01198-5.
  11. ↑ Grant-Kels JM (2007). Color Atlas of Dermatopathology (Dermatology: Clinical & Basic Science). Informa Healthcare. pp. 163. ISBN 0-8493-3794-1.
  12. ↑ Ryan, T (1991). "Cutaneous Circulation". In Goldsmith, Lowell A. Physiology, biochemistry, and molecular biology of the skin (2nd ed.). New York: Oxford University Press. p. 1019. ISBN 0-19-505612-4.
  13. ↑ Swerlick RA, Lawley TJ; Lawley (January 1993). "Role of microvascular endothelial cells in inflammation". J. Invest. Dermatol. 100 (1): 111S–115S. doi:10.1038/jid.1993.33. PMID 8423379.
  14. 1 2 3 4 Lynch, Peter J (1994). Dermatology. Williams & Wilkins. ISBN 0-683-05252-7.
  15. ↑ King, LS (1954). "What Is Disease?". Philosophy of Science. 21 (3): 193–203. doi:10.1086/287343.
  16. 1 2 Bluefarb, Samuel M (1984). Dermatology. Upjohn Co. ISBN 0-89501-004-6.
  17. ↑ Tilles G, Wallach D; Wallach (1989). "[The history of nosology in dermatology]". Ann Dermatol Venereol (in French). 116 (1): 9–26. PMID 2653160.
  18. ↑ Lambert WC, Everett MA; Everett (October 1981). "The nosology of parapsoriasis". J. Am. Acad. Dermatol. 5 (4): 373–95. doi:10.1016/S0190-9622(81)70100-2. PMID 7026622.
  19. ↑ Jackson R (1977). "Historical outline of attempts to classify skin diseases". Can Med Assoc J. 116 (10): 1165–68. PMC 1879511Freely accessible. PMID 324589.
  20. ↑ Copeman PW (February 1995). "The creation of global dermatology". J R Soc Med. 88 (2): 78–84. PMC 1295100Freely accessible. PMID 7769599.
  21. ↑ Fitzpatrick, Thomas B; Klauss Wolff; Wolff, Klaus Dieter; Johnson, Richard R.; Suurmond, Dick; Richard Suurmond (2005). Fitzpatrick's color atlas and synopsis of clinical dermatology. McGraw-Hill Medical Pub. Division. ISBN 0-07-144019-4.
  22. ↑ Werner B (August 2009). "[Skin biopsy and its histopathologic analysis: Why? What for? How? Part I]". An Bras Dermatol (in Portuguese). 84 (4): 391–5. PMID 19851671.
  23. ↑ Werner B (October 2009). "[Skin biopsy with histopathologic analysis: why? what for? how? part II]". An Bras Dermatol (in Portuguese). 84 (5): 507–13. doi:10.1590/S0365-05962009000500010. PMID 20098854.
  24. ↑ Xiaowei Xu; Elder, David A; Rosalie Elenitsas; Johnson, Bernett L; Murphy, George E (2008). Lever's Histopathology of the Skin. Hagerstwon, MD: Lippincott Williams & Wilkins. ISBN 0-7817-7363-6.
  25. ↑ Weedon's Skin Pathology, 2-Volume Set: Expert Consult - Online and Print. Edinburgh: Churchill Livingstone. 2009. ISBN 0-7020-3941-1.
  26. 1 2 3 4 5 David J DiCaudo; Dirk Elston MD; Dirk M Elston; Tammie Ferringer; Christine J Ko; Christine Ko MD; Steven Peckham; Whitney A High (2009). Dermatopathology. Philadelphia: Saunders. ISBN 0-7020-3023-6.
  27. ↑ Rustin MH (1990). "Dermatology". Postgrad Med J. 66 (781): 894–905. doi:10.1136/pgmj.66.781.894. PMC 2429766Freely accessible. PMID 2148371.
  28. ↑ Feldman S, Careccia RE, Barham KL, Hancox J; Careccia; Barham; Hancox (2004). "Diagnosis and treatment of acne". Am Fam Physician. 69 (9): 2123–30. PMID 15152959.
  29. ↑ Fietta P (2004). "Autoinflammatory diseases: the hereditary periodic fever syndromes". Acta Biomed. 75 (2): 92–9. PMID 15481697.
  30. ↑ Centola M, Aksentijevich I, Kastner DL; Aksentijevich; Kastner (1998). "The hereditary periodic fever syndromes: molecular analysis of a new family of inflammatory diseases". Hum Mol Genet. 7 (10): 1581–8. doi:10.1093/hmg/7.10.1581. PMID 9735379.
  31. ↑ Chan, Lawrence S (30 March 2009). Blistering Skin Diseases (1 ed.). Manson Publishing Ltd. ISBN 978-1-84076-066-8.
  32. ↑ Yeh SW, Ahmed B, Sami N, Razzaque Ahmed A; Ahmed; Sami; Razzaque Ahmed (2003). "Blistering disorders: diagnosis and treatment". Dermatol Ther. 16 (3): 214–23. doi:10.1046/j.1529-8019.2003.01631.x. PMID 14510878.
  33. ↑ Eming R, Hertl M; Hertl; Autoimmune Diagnostics Working Group (2006). "Autoimmune bullous disorders". Clin Chem Lab Med. 44 (2): 144–9. doi:10.1515/CCLM.2006.027. PMID 16475898.
  34. 1 2 3 4 James, William D; et al. (2006). Andrews' Diseases of the Skin: Clinical Dermatology. Saunders Elsevier. ISBN 0-7216-2921-0.
  35. 1 2 3 4 5 Christine J Ko; Schwarzenberger, Kathryn; Werchniak, Andrew E (2009). General dermatology. Philadelphia: Saunders. ISBN 0-7020-3093-7.
  36. ↑ Aronson IK, Tharp MD; Worobec (2010). "Diagnosis and Treatment of Panniculitis". Dermatologic Therapy. 23 (4): 317–434. doi:10.1111/j.1529-8019.2010.01331.x. ISSN 1529-8019.
  37. ↑ Requena L, Yus ES; Yus (2001). "Panniculitis. Part I. Mostly septal panniculitis". J Am Acad Dermatol. 45 (2): 163–83; quiz 184–6. doi:10.1067/mjd.2001.114736. PMID 11464178.
  38. ↑ Requena L, Sánchez Yus E; Sánchez Yus (2001). "Panniculitis. Part II. Mostly lobular panniculitis". J Am Acad Dermatol. 45 (3): 325–61; quiz 362–64. doi:10.1067/mjd.2001.114735. PMID 11511831.
  39. ↑ Phelps RG, Shoji T; Shoji (2001). "Update on panniculitis". Mt Sinai J Med. 68 (4–5): 262–7. PMID 11514913.
  40. ↑ Küster W, Traupe H; Traupe (September 1988). "[Clinical aspects and genetics of congenital skin defects]". Hautarzt (in German). 39 (9): 553–63. PMID 3053531.
  41. ↑ Andrea L. Zaenglein; MD, Howard Gimbel; Albert C Yan (2008). Pediatric Dermatology: Requisites in Dermatology. Saunders Ltd. ISBN 0-7020-3022-8.
  42. ↑ Crofford, Leslie J; Klippel, John H; Weyand, Cornelia M; Stone, John F (2001). Primer on the rheumatic diseases. Atlanta, GA: Arthritis Foundation. ISBN 0-912423-29-3.
  43. ↑ Ushiki T (2002). "Collagen fibers, reticular fibers and elastic fibers. A comprehensive understanding from a morphological viewpoint" (PDF). Arch Histol Cytol. 65 (2): 109–26. doi:10.1679/aohc.65.109. PMID 12164335.
  44. ↑ Alsaad KO, Ghazarian D; Ghazarian (2005). "My approach to superficial inflammatory dermatoses". J Clin Pathol. 58 (12): 1233–41. doi:10.1136/jcp.2005.027151. PMC 1770784Freely accessible. PMID 16311340.
  45. ↑ Wüthrich B, Cozzio A, Roll A, Senti G, Kündig T, Schmid-Grendelmeier P; Cozzio; Roll; Senti; Kündig; Schmid-Grendelmeier (2007). "Atopic eczema: genetics or environment?". Ann Agric Environ Med. 14 (2): 195–201. PMID 18247450.
  46. ↑ Roosterman D, Goerge T, Schneider SW, Bunnett NW, Steinhoff M; Goerge; Schneider; Bunnett; Steinhoff (2006). "Neuronal control of skin function: the skin as a neuroimmunoendocrine organ". Physiol Rev. 86 (4): 1309–79. doi:10.1152/physrev.00026.2005. PMID 17015491.
  47. ↑ Spergel JM (August 2010). "From atopic dermatitis to asthma: the atopic march". Ann. Allergy Asthma Immunol. 105 (2): 99–106; quiz 107–9, 117. doi:10.1016/j.anai.2009.10.002. PMID 20674819.
  48. ↑ Saint-Mezard P, Rosieres A, Krasteva M, et al. (2004). "Allergic contact dermatitis". Eur J Dermatol. 14 (5): 284–95. PMID 15358566.
  49. ↑ Krasteva M, Kehren J, Sayag M, et al. (1999). "Contact dermatitis II. Clinical aspects and diagnosis". Eur J Dermatol. 9 (2): 144–59. PMID 10066966.
  50. ↑ Sharma VK, Asati DP; Asati (2010). "Pediatric contact dermatitis". Indian J Dermatol Venereol Leprol. 76 (5): 514–20. doi:10.4103/0378-6323.69070. PMID 20826990.
  51. ↑ Buxton PK (1987). "ABC of dermatology. Eczema and dermatitis". Br Med J (Clin Res Ed). 295 (6605): 1048–51. doi:10.1136/bmj.295.6605.1048. PMC 1248082Freely accessible. PMID 3120868.
  52. ↑ Mengesha YM, Bennett ML; Bennett (2002). "Pustular skin disorders: diagnosis and treatment". Am J Clin Dermatol. 3 (6): 389–400. doi:10.2165/00128071-200203060-00003. PMID 12113648.
  53. ↑ Schwartz RA, Janusz CA, Janniger CK; Janusz; Janniger (2006). "Seborrheic dermatitis: an overview". Am Fam Physician. 74 (1): 125–30. PMID 16848386.
  54. ↑ Costin GE, Hearing VJ; Hearing (2007). "Human skin pigmentation: melanocytes modulate skin color in response to stress". FASEB J. 21 (4): 976–94. doi:10.1096/fj.06-6649rev. PMID 17242160.
  55. ↑ Yamaguchi Y, Brenner M, Hearing VJ; Brenner; Hearing (2007). "The regulation of skin pigmentation". J Biol Chem. 282 (38): 27557–61. doi:10.1074/jbc.R700026200. PMID 17635904.
  56. ↑ Davis EC, Callender VD; Callender (July 2010). "Postinflammatory hyperpigmentation: a review of the epidemiology, clinical features, and treatment options in skin of color". J Clin Aesthet Dermatol. 3 (7): 20–31. PMC 2921758Freely accessible. PMID 20725554.
  57. ↑ Goolamali SK (1985). "Drug eruptions". Postgrad Med J. 61 (720): 925–33. doi:10.1136/pgmj.61.720.925. PMC 2418295Freely accessible. PMID 2932688.
  58. ↑ Svensson CK, Cowen EW, Gaspari AA; Cowen; Gaspari (2001). "Cutaneous drug reactions". Pharmacol Rev. 53 (3): 357–79. PMID 11546834.
  59. ↑ Ramdial PK, Naidoo DK; Naidoo (June 2009). "Drug-induced cutaneous pathology". J. Clin. Pathol. 62 (6): 493–504. doi:10.1136/jcp.2008.058289. PMID 19155238.
  60. ↑ Reichrath J (July 2009). "The skin is a fascinating endocrine organ". Dermatoendocrinol. 1 (4): 195–6. doi:10.4161/derm.1.4.9653. PMC 2835874Freely accessible. PMID 20592790.
  61. ↑ Slominski A, Wortsman J; Wortsman (2000). "Neuroendocrinology of the skin". Endocr Rev. 21 (5): 457–87. doi:10.1210/er.21.5.457. PMID 11041445.
  62. ↑ Heymann WR (July 2006). "Eosinophilic dermatoses". J. Am. Acad. Dermatol. 55 (1): 114–5. doi:10.1016/j.jaad.2006.02.058. PMID 16781301.
  63. ↑ Machado-Pinto J, McCalmont TH, Golitz LE; McCalmont; Golitz (December 1996). "Eosinophilic and neutrophilic spongiosis: clues to the diagnosis of immunobullous diseases and other inflammatory disorders". Semin Cutan Med Surg. 15 (4): 308–16. doi:10.1016/S1085-5629(96)80044-7. PMID 9069598.
  64. ↑ Joel L. Spitz (2005). Genodermatoses: a clinical guide to genetic skin disorders. Lippincott Williams & Wilkins. ISBN 0-7817-4088-6.
  65. ↑ McLean WH; Epithelial Genetics, Group (2003). "Genetic disorders of palm skin and nail". J Anat. 202 (1): 133–41. doi:10.1046/j.1469-7580.2003.00141.x. PMC 1571049Freely accessible. PMID 12587928.
  66. 1 2 3 4 5 6 Habif, Thomas P. (2001). Skin disease: diagnosis and treatment. Mosby. ISBN 0-8151-3762-1.
  67. ↑ Stulberg DL, Penrod MA, Blatny RA; Penrod; Blatny (2002). "Common bacterial skin infections". Am Fam Physician. 66 (1): 119–24. PMID 12126026.
  68. ↑ Frankel A, Penrose C, Emer J; Penrose; Emer (October 2009). "Cutaneous tuberculosis: a practical case report and review for the dermatologist". J Clin Aesthet Dermatol. 2 (10): 19–27. PMC 2923933Freely accessible. PMID 20725570.
  69. ↑ Diaz JH (2010). "Mite-transmitted dermatoses and infectious diseases in returning travelers". J Travel Med. 17 (1): 21–31. doi:10.1111/j.1708-8305.2009.00352.x. PMID 20074098.
  70. ↑ Lebwohl MG, Rosen, Stockfleth (November 2010). "The role of human papillomavirus in common skin conditions: current viewpoints and therapeutic options". Cutis. 86 (5): suppl 1–11; quiz suppl 12. PMID 21214125.
  71. ↑ Ismail SB, Kumar SK, Zain RB; Kumar; Zain (2007). "Oral lichen planus and lichenoid reactions: etiopathogenesis, diagnosis, management and malignant transformation" (PDF). J Oral Sci. 49 (2): 89–106. doi:10.2334/josnusd.49.89. PMID 17634721.
  72. ↑ Katta R (2000). "Lichen planus". Am Fam Physician. 61 (11): 3319–24, 3327–8. PMID 10865927.
  73. ↑ Kanwar AJ, De D; De (2010). "Lichen planus in children". Indian J Dermatol Venereol Leprol. 76 (4): 366–72. doi:10.4103/0378-6323.66581. PMID 20657116.
  74. ↑ Connors JM, Hsi ED, Foss FM; Hsi; Foss (2002). "Lymphoma of the skin". Hematology Am Soc Hematol Educ Program. 2002 (1): 263–82. doi:10.1182/asheducation-2002.1.263. PMID 12446427.
  75. ↑ Alan N Houghton; Balch, Charles M. (1992). Cutaneous melanoma. J.B. Lippincott. ISBN 0-397-51052-7.
  76. ↑ Venkatesan A (October 2010). "Pigmented lesions of the vulva". Dermatol Clin. 28 (4): 795–805. doi:10.1016/j.det.2010.08.007. PMID 20883921.
  77. ↑ Petrescu I, Condrea C, Alexandru A, et al. (2010). "Diagnosis and treatment protocols of cutaneous melanoma: latest approach 2010". Chirurgia (Bucur). 105 (5): 637–43. PMID 21141087.
  78. ↑ Wojas-Pelc A, Rajzer L, Jaworek A, Woźniak W; Rajzer; Jaworek; Woźniak (2006). "[The latest diagnostical methods and therapy in melanoma]". Prz. Lek. (in Polish). 63 (8): 674–80. PMID 17441381.
  79. ↑ Krone B, Grange JM; Grange (December 2010). "Melanoma, Darwinian medicine and the inner world". J. Cancer Res. Clin. Oncol. 136 (12): 1787–94. doi:10.1007/s00432-010-0949-x. PMC 2962785Freely accessible. PMID 20852885.
  80. ↑ Hawryluk EB, Izikson L, English JC; Izikson; English Jc (2010). "Non-Infectious Granulomatous Diseases of the Skin and their Associated Systemic Diseases: An Evidence-Based Update to Important Clinical Questions". Am J Clin Dermatol. 11 (3): 171–81. doi:10.2165/11530080-000000000-00000. PMID 20184390.
  81. ↑ Vardiman JW (March 2010). "The World Health Organization (WHO) classification of tumors of the hematopoietic and lymphoid tissues: an overview with emphasis on the myeloid neoplasms". Chem. Biol. Interact. 184 (1–2): 16–20. doi:10.1016/j.cbi.2009.10.009. PMID 19857474.
  82. ↑ Dempsey OJ, Paterson EW, Kerr KM, Denison AR; Paterson; Kerr; Denison (2009). "Sarcoidosis". BMJ. 339: b3206. doi:10.1136/bmj.b3206. PMID 19717499.
  83. ↑ Jafferany M (2007). "Psychodermatology: a guide to understanding common psychocutaneous disorders". Prim Care Companion J Clin Psychiatry. 9 (3): 203–13. doi:10.4088/pcc.v09n0306. PMC 1911167Freely accessible. PMID 17632653.
  84. ↑ Harth W, Hermes B, Niemeier V, Gieler U; Hermes; Niemeier; Gieler (2006). "Clinical pictures and classification of somatoform disorders in dermatology". Eur J Dermatol. 16 (6): 607–14. PMID 17229599.
  85. ↑ Sillevis Smitt JH, Wulffraat NM, Kuijpers TW; Wulffraat; Kuijpers (2005). "The skin in primary immunodeficiency disorders". Eur J Dermatol. 15 (6): 425–32. PMID 16280293.
  86. ↑ Cooper MD, Lanier LL, Conley ME, Puck JM; Lanier; Conley; Puck (2003). "Immunodeficiency disorders". Hematology Am Soc Hematol Educ Program. 2003 (1): 314–30. doi:10.1182/asheducation-2003.1.314. PMID 14633788.
  87. ↑ Miller SJ (1989). "Nutritional deficiency and the skin". J Am Acad Dermatol. 21 (1): 1–30. doi:10.1016/S0190-9622(89)70144-4. PMID 2663932.
  88. ↑ Heath ML, Sidbury R; Sidbury (2006). "Cutaneous manifestations of nutritional deficiency". Curr Opin Pediatr. 18 (4): 417–22. doi:10.1097/01.mop.0000236392.87203.cc. PMID 16914997.
  89. ↑ Christiano AM (June 1997). "Frontiers in keratodermas: pushing the envelope". Trends Genet. 13 (6): 227–33. doi:10.1016/S0168-9525(97)01104-9. PMID 9196328.
  90. ↑ Shornick JK (September 1998). "Dermatoses of pregnancy". Semin Cutan Med Surg. 17 (3): 172–81. doi:10.1016/S1085-5629(98)80011-4. PMID 9759674.
  91. ↑ Holmes RC, Black MM; Black (March 1983). "The specific dermatoses of pregnancy". J. Am. Acad. Dermatol. 8 (3): 405–12. doi:10.1016/S0190-9622(83)70046-0. PMID 6833540.
  92. ↑ Greaves MW (2007). "Recent advances in pathophysiology and current management of itch" (PDF). Ann Acad Med Singap. 36 (9): 788–92. PMID 17925991.
  93. ↑ Steinhoff M, Bienenstock J, Schmelz M, Maurer M, Wei E, Bíró T; Bienenstock; Schmelz; Maurer; Wei; Bíró (2006). "Neurophysiological, neuroimmunological, and neuroendocrine basis of pruritus". J Invest Dermatol. 126 (8): 1705–18. doi:10.1038/sj.jid.5700231. PMID 16845410.
  94. ↑ Langley RG, Krueger GG, Griffiths CE; Krueger; Griffiths (2005). "Psoriasis: epidemiology, clinical features, and quality of life". Ann Rheum Dis. 64 (Suppl 2): ii18–23; discussion ii24–5. doi:10.1136/ard.2004.033217. PMC 1766861Freely accessible. PMID 15708928.
  95. ↑ Luba KM, Stulberg DL; Stulberg (2006). "Chronic plaque psoriasis". Am Fam Physician. 73 (4): 636–44. PMID 16506705.
  96. ↑ Krueger JG, Bowcock A; Bowcock (2005). "Psoriasis pathophysiology: current concepts of pathogenesis". Ann Rheum Dis. 64 (Suppl 2): ii30–6. doi:10.1136/ard.2004.031120. PMC 1766865Freely accessible. PMID 15708932.
  97. ↑ Callen JP (2002). "Neutrophilic dermatoses". Dermatol Clin. 20 (3): 409–19. doi:10.1016/S0733-8635(02)00006-2. PMID 12170875.
  98. ↑ Wallach D, Vignon-Pennamen MD; Vignon-Pennamen (2006). "From acute febrile neutrophilic dermatosis to neutrophilic disease: forty years of clinical research". J Am Acad Dermatol. 55 (6): 1066–71. doi:10.1016/j.jaad.2006.07.016. PMID 17097401.
  99. ↑ Ermertcan AT, Ertan P; Ertan (2010). "Skin manifestations of child abuse". Indian J Dermatol Venereol Leprol. 76 (4): 317–26. doi:10.4103/0378-6323.66572. PMID 20657110.
  100. ↑ Rebecca Tung; Murad Alam MD; Hayes B Gladstone (2008). Cosmetic Dermatology: Requisites in Dermatology Series. Saunders Ltd. ISBN 0-7020-3143-7.
  101. ↑ Hymes SR, Strom EA, Fife C; Strom; Fife (January 2006). "Radiation dermatitis: clinical presentation, pathophysiology, and treatment 2006". J. Am. Acad. Dermatol. 54 (1): 28–46. doi:10.1016/j.jaad.2005.08.054. PMID 16384753.
  102. ↑ Clarke P (2004). "Urticaria". Aust Fam Physician. 33 (7): 501–03. PMID 15301166.
  103. ↑ Muller BA (2004). "Urticaria and angioedema: a practical approach". Am Fam Physician. 69 (5): 1123–8. PMID 15023012.
  104. ↑ Carlson JA, Cavaliere LF, Grant-Kels JM; Cavaliere; Grant-Kels (2006). "Cutaneous vasculitis: diagnosis and management". Clin. Dermatol. 24 (5): 414–29. doi:10.1016/j.clindermatol.2006.07.007. PMID 16966021.
  105. ↑ Panuncialman J, Falanga V; Falanga (December 2010). "Unusual causes of cutaneous ulceration". Surg. Clin. North Am. 90 (6): 1161–80. doi:10.1016/j.suc.2010.08.006. PMC 2991050Freely accessible. PMID 21074034.

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